Our latest experiments reveal this is due to defective trafficking of late endosomes to lysosomes, with concomitant homotypic fusion with the influenced vesicular compartments [five]. The defect in lysosome-directed trafficking also affects autophagic flux, with resultant accumulation of autophagosomes [5]. In the long run, the integrity of your cell membrane https://donaldq530ejm2.atualblog.com/profile